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Commentary
Animal models for Gaucher disease research
Tamar Farfel-Becker, Einat B. Vitner, Anthony H. Futerman
Disease Models & Mechanisms 2011 4: 746-752; doi: 10.1242/dmm.008185
Tamar Farfel-Becker
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Einat B. Vitner
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Anthony H. Futerman
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  • For correspondence: tony.futerman@weizmann.ac.il
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Article Information

vol. 4 no. 6, 746-752

DOI 
https://doi.org/10.1242/dmm.008185
PubMed 
21954067

Published By 
The Company of Biologists Ltd
Print ISSN 
1754-8403
Online ISSN 
1754-8411
History 
  • Published online November 7, 2011.
Posted online 
September 27, 2011
Copyright & Usage 
© 2011. Published by The Company of Biologists Ltd This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial Share Alike License (http://creativecommons.org/licenses/by-nc-sa/3.0), which permits unrestricted non-commercial use, distribution and reproduction in any medium provided that the original work is properly cited and all further distributions of the work or adaptation are subject to the same Creative Commons License terms.

Article Versions

  • Previous version (September 27, 2011 - 00:43).
  • You are viewing the most recent version of this article.

Author Information

  1. Tamar Farfel-Becker1,*,
  2. Einat B. Vitner1,* and
  3. Anthony H. Futerman1,‡
  1. 1Department of Biological Chemistry, Weizmann Institute of Science, Rehovot 76100, Israel
  1. ↵‡ Author for correspondence (tony.futerman{at}weizmann.ac.il)
  1. ↵* These authors contributed equally to this work

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Commentary
Animal models for Gaucher disease research
Tamar Farfel-Becker, Einat B. Vitner, Anthony H. Futerman
Disease Models & Mechanisms 2011 4: 746-752; doi: 10.1242/dmm.008185
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Commentary
Animal models for Gaucher disease research
Tamar Farfel-Becker, Einat B. Vitner, Anthony H. Futerman
Disease Models & Mechanisms 2011 4: 746-752; doi: 10.1242/dmm.008185

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  • Top
  • Article
    • Abstract
    • Introduction
    • Chemically induced models of GD
    • The Gba-knockout mouse
    • Mouse models of GD based on point mutations
    • RecNciI and L444P/L444P mice
    • N370S, V394L, D409H and D409V point mutations
    • Conditional GD mice
    • Non-mouse animal models
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