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Preprint Highlights
The latest from the preLights community – March 2020
Disease Models & Mechanisms 2020 13: p0301
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Loss of epithelial polarity redirects Notch signaling and triggers a Xrp1 response during neoplastic growth in Drosophila by Logeay et al.

Invasive Procedure: new insights into how changes in epithelial structure can turn Notch nasty.

Selected by Sally Lowell. Read the guest preLight here

 

The WD and linker domains of ATG16L1 required for non-canonical autophagy limit lethal influenza A virus infection at epithelial surfaces by Wang et al.

Loss of non-canonical autophagy in mice increases sensitivity to influenza A virus infection by disrupting defences at the lung epithelial barrier, but not in phagocytic leukocytes.

Selected by Kirsty Hooper. Read the preLight here

 

Bariatric surgery reveals a gut-restricted TGR5 agonist that exhibits anti-diabetic effects by Chaudhari et al.

A gut check for bariatric surgery – Surgery-mediated bile acid changes improve metabolic parameters.

Selected by Connor Rosen. Read the preLight here

 

NINJA: an inducible genetic model for creating neoantigens in vivo by Damo et al.

A neoantigen in disguise, the NINJA system: testing the functionality of a novel genetically engineered mouse model with an inducible neoantigen construct.

Selected by Deborah Caswell. Read the preLight here

 

Obesity-driven changes in ECM composition promote local invasion and metastasis of breast tumors by Wishart et al.

Obesity and breast cancer – Collagen VI in the spotlight

Selected by Vassilis Papalazarou and Anh Hoang Le. Read the preLight here

 

Regeneration in the adult Drosophila brain by Crocker et al.

A hidden stem cell-like population found in the Drosophila brain: let's regenerate.

Selected by Ivana Viktorinová. Read the preLight here

 

On-demand spatiotemporal programming of collective cell migration via bioelectric stimulation by Zajdel et al.

An electricity-based pied piper of Hamelin: moving cells on demand.

Selected by Mariana De Niz. Read the preLight here

 

Mutational signatures are jointly shaped by DNA damage and repair by Volkova et al.

A combinatory mutational screen in C. elegans, combining 54 DNA repair mutants and 12 genotoxins, demonstrates that mutations are a result of both DNA damage and failed repair.

Selected by Kerryn Elliott. Read the preLight here

 

Chemogenetic analysis of how receptors for short chain fatty acids regulate the gut-brain axis by Barki et al.

Following your gut – using chemogenetics to understand how short chain fatty acids regulate the gut-brain axis.

Selected by Phyllis Phuah. Read the preLight here

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DMM and COVID-19

We are aware that the COVID-19 pandemic is having an unprecedented impact on researchers worldwide. The Editors of all The Company of Biologists’ journals have been considering ways in which we can alleviate concerns that members of our community may have around publishing activities during this time. Read about the actions we are taking at this time.

Please don’t hesitate to contact the Editorial Office if you have any questions or concerns.


The twin pillars of Disease Models & Mechanisms

In her first Editorial as Editor-in-Chief, Liz Patton sets out her vision and priorities for DMM focusing on four thematic challenges: mechanisms of disease, innovative technologies, disease progression through time and therapy.


Extended deadline - The RAS Pathway: Diseases, Therapeutics and Beyond

Our upcoming special issue is welcoming submissions until 3 May 2021. Guest-edited by Donita Brady (Perelman School of Medicine at the University of Pennsylvania, USA) and Arvin Dar (Icahn School of Medicine at Mount Sinai, USA), the issue will focus on the targeting the RAS pathway.

Find out more about the issue and how to submit your manuscript.


Perspective - Modelling the developmental origins of paediatric cancer to improve patient outcomes

James Amatruda authors our first Perspective, discussing some of the key challenges in paediatric cancer from his perspective as a physician-scientist.


A muscle growth-promoting treatment based on the attenuation of activin/myostatin signalling results in long-term testicular abnormalities

In this issue’s Editor’s choice, Ketan Patel and colleagues describe how even brief exposure to muscle-growth-promoting treatments exerts a long-term detrimental effect on the testes, and test promising therapeutics to mitigate this side-effect.

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