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Table of Contents

February 1, 2015; 8 (2)

IN THIS ISSUE

  • Open Access
    Vitamin A dimers as primary drivers of retinal degeneration
    Disease Models & Mechanisms 2015 8: e0201
  • Open Access
    Effects of maternal diabetes on the Wnt-PCP pathway and embryogenesis
    Disease Models & Mechanisms 2015 8: e0202

A MODEL FOR LIFE

  • Open Access
    An odyssey in the space of molecules, genes, biology and brain: an interview with Sabine Cordes
    Disease Models & Mechanisms 2015 8: 105-108; doi: 10.1242/dmm.019893

REVIEW

  • Open Access
    Biology and therapy of inherited retinal degenerative disease: insights from mouse models
    Shobi Veleri, Csilla H. Lazar, Bo Chang, Paul A. Sieving, Eyal Banin, Anand Swaroop
    Disease Models & Mechanisms 2015 8: 109-129; doi: 10.1242/dmm.017913

RESEARCH ARTICLES

  • Open Access
    Morphological and physiological retinal degeneration induced by intravenous delivery of vitamin A dimers in rabbits
    Jackie Penn, Doina M. Mihai, Ilyas Washington
    Disease Models & Mechanisms 2015 8: 131-138; doi: 10.1242/dmm.017194
  • Open Access
    Impact of retinoic acid exposure on midfacial shape variation and manifestation of holoprosencephaly in Twsg1 mutant mice
    Charles J. Billington, Jr, Brian Schmidt, Ralph S. Marcucio, Benedikt Hallgrimsson, Rajaram Gopalakrishnan, Anna Petryk
    Disease Models & Mechanisms 2015 8: 139-146; doi: 10.1242/dmm.018275
  • Open Access
    Partial genetic suppression of a loss-of-function mutant of the neuronal ceroid lipofuscinosis-associated protease TPP1 in Dictyostelium discoideum
    Jonathan E. Phillips, Richard H. Gomer
    Disease Models & Mechanisms 2015 8: 147-156; doi: 10.1242/dmm.018820
  • Open Access
    The effect of maternal diabetes on the Wnt-PCP pathway during embryogenesis as reflected in the developing mouse eye
    Beatriz López-Escobar, David A. Cano, Anabel Rojas, Beatriz de Felipe, Francisco Palma, José A. Sánchez-Alcázar, Deborah Henderson, Patricia Ybot-González
    Disease Models & Mechanisms 2015 8: 157-168; doi: 10.1242/dmm.017723
  • Open Access
    HIF-1α restricts NF-κB-dependent gene expression to control innate immunity signals
    Daniel Bandarra, John Biddlestone, Sharon Mudie, H.-Arno J. Müller, Sonia Rocha
    Disease Models & Mechanisms 2015 8: 169-181; doi: 10.1242/dmm.017285
  • Open Access
    In vitro treatment of HepG2 cells with saturated fatty acids reproduces mitochondrial dysfunction found in nonalcoholic steatohepatitis
    Inmaculada García-Ruiz, Pablo Solís-Muñoz, Daniel Fernández-Moreira, Teresa Muñoz-Yagüe, José A. Solís-Herruzo
    Disease Models & Mechanisms 2015 8: 183-191; doi: 10.1242/dmm.018234
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  • Open Access

    Altered expression of the Cdk5 activator-like protein, Cdk5α, causes neurodegeneration, in part by accelerating the rate of aging

    Joshua Spurrier, Arvind Kumar Shukla, Kristina McLinden, Kory Johnson, Edward Giniger
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  • Open Access

    Noncanonical NF-κB signaling and the essential kinase NIK modulate crucial features associated with eosinophilic esophagitis pathogenesis

    Kristin Eden, Daniel E. Rothschild, Dylan K. McDaniel, Bettina Heid, Irving C. Allen
    PDF
  • Open Access

    Modelling amyotrophic lateral sclerosis: progress and possibilities

    Philip Van Damme, Wim Robberecht, Ludo Van Den Bosch
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  • Open Access

    Modelling glioblastoma tumour-host cell interactions using adult brain organotypic slice co-culture

    Maria Angeles Marques-Torrejon, Ester Gangoso, Steven M. Pollard
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    Use of zebrafish to study Shigella infection

    Gina M. Duggan, Serge Mostowy
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Editor’s Choice – Altered expression of the Cdk5 activator-like protein, Cdk5α, causes neurodegeneration, in part by accelerating the rate of aging

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DMM aims to promote human health by encouraging collaboration between basic and clinical researchers, covering a diverse range of diseases, approaches and models. Our Editors are all active researchers in the field – your peers, colleagues and mentors, who know how much work has gone into every paper. Recently, we have introduced format-free submission, and we accept peer review reports from other journals, making submission as easy as possible for our authors. Send us your next great paper – publish with us and you'll be in good company.

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